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Science Forum Index » Medicine - Cancer Forum » Soy and fish protect from cancer: study
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| Roman Bystrianyk |
 Posted: Wed Nov 15, 2006 8:55 am |
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"Soy and fish protect from cancer: study", Reuters, November 14, 2006,
Link:
http://today.reuters.com/news/articlenews.aspx?type=healthNews&storyID=2006-11-14T194442Z_01_N14227042_RTRUKOC_0_US-CANCER-DIET.xml&WTmodLoc=NewsHome-C3-healthNews-3
People who ate soy regularly as children have a lower risk of breast
cancer, researchers reported on Tuesday.
And men who eat fish several times a week have a lower risk of colon
cancer, a second team of researchers told a meeting in Boston of the
American Association for Cancer Research.
The studies add to a growing body of evidence about the role of diet in
cancer. Cancer experts now believe that up to two-thirds of all cancers
come from lifestyle factors such as smoking, diet and lack of exercise.
Dr. Larissa Korde of the National Cancer Institute and colleagues at
the University of Hawaii studied studied 597 Asian-American women with
breast cancer and 966 women without the disease. The mothers of some of
the women were also available to answer questions about what they fed
their daughters as children.
The women who ate the most soy-based foods such as tofu and miso when
aged 5 to 11 reduced their risk of developing breast cancer by 58
percent, the researchers found.
"Childhood soy intake was significantly associated with reduced breast
cancer risk in our study, suggesting that the timing of soy intake may
be especially critical," Korde said.
It is not clear how soy might prevent cancer, although compounds in soy
called isoflavones have estrogen-like effects.
A second study presented at the same meeting showed that men who ate
fish five times a week or more had a 40 percent lower risk of
developing colorectal cancer compared to men who ate fish less than
once a week.
Megan Phillips of the Harvard School of Public Health and colleagues
analyzed data from 22,071 men volunteering for a large, ongoing study
of physicians.
"We already know that eating fish can reduce the risk of sudden cardiac
death, and this might provide another reason to add fish to your diet,"
said Megan Phillips, a doctoral student at the Harvard School of Public
Health, who led the study.
Many kinds of fish are rich in omega-3 fatty acids, which in turn
interfere with the cyclooxygenase-2 or COX-2 enzyme.
COX-2 affects inflammation, which may play a role in tumor growth.
Men who ate the most fish had a 40 percent lower risk of developing
colorectal cancer over the next 19 years, the researchers found. Eating
fish twice a week lowered the risk by 13 percent. Aspirin did not add
any extra benefit or risk.
A separate study published on Monday found that eating red meat raised
the risk of breast cancer in women. |
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| Guest |
| Posted: Thu Nov 16, 2006 1:37 am |
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Wouldn't it be nice if they informed the public that a diet very low in
unsaturated fatty acids will result in no COX- 2 activity?
And wouldn't it be nice if they told the public that studies about
things like soy and fish only make these items look healthy because
they are being substituted for items that are even more unhealthy?
Oh, and wouldn't it be nice if they made the public aware of
populations that consumed diets that should be unhealthy, according to
their dogma, but are nearly free of "chronic diseases?"
When that day dawns, I may give some consideration to these kinds of
studies. |
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| Vernon |
| Posted: Thu Nov 16, 2006 10:14 am |
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<monty1945@lycos.com> wrote in message
news:1163655429.885027.41540@m7g2000cwm.googlegroups.com...
Quote: Wouldn't it be nice if they informed the public that a diet very low in
unsaturated fatty acids will result in no COX- 2 activity?
And wouldn't it be nice if they told the public that studies about
things like soy and fish only make these items look healthy because
they are being substituted for items that are even more unhealthy?
Oh, and wouldn't it be nice if they made the public aware of
populations that consumed diets that should be unhealthy, according to
their dogma, but are nearly free of "chronic diseases?"
When that day dawns, I may give some consideration to these kinds of
studies.
Are "they" out to get you? |
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| Ron Peterson |
| Posted: Thu Nov 16, 2006 1:12 pm |
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monty1945@lycos.com wrote:
Quote: Wouldn't it be nice if they informed the public that a diet very low in
unsaturated fatty acids will result in no COX- 2 activity?
COX-2 is essential for fracture healing, so it may not be desirable to
suppress COX-2 activity.
--
Ron |
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| Guest |
| Posted: Thu Nov 16, 2006 2:49 pm |
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Quote: COX-2 is essential for fracture healing, so it may not be desirable to
suppress COX-2 activity.
Care to cite some on point evidence for this claim, Ron?
You do understand that Mead acid cannot be made into COX-2 metabolites,
right?
The 1948 experiment found that rats had no problems on a fat free diet,
one that would also mean no COX-2 activity. I have done this
experiment to myself since 2001, though I eat highly saturated fats
with very little linoleic acid content. I notice that cuts actually
heal much better than before, with no scarring problems.
I will challenge you to an experiment in which rats are fed two
different diets, one that is 30% canola and fish oil, and another that
is 30% coconut oil (my choice). If there is no discernable difference
in healing, or if the coconut fed rats heal better, then you pay for
all expenses. Note that biases must be discarded. It is true that the
AA metabolites are more reactive, and cuts might heal a bit faster, but
this often means scarring, and it has nothing to do with claims of
"essentiality."
As to the use of the term "they." Since just about all the "experts"
in the USA are making these kinds of claims, how could I name them all?
I could say, "everyone except Ray Peat and a few others," but I doubt
that you would be satisfied by this qualification, or would you? The
evidence requires no conspiracy theory, only an advocate who is not
conflicted and who is willing to examine it without any biases. |
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| Vernon |
| Posted: Thu Nov 16, 2006 4:41 pm |
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Guest
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"Ron Peterson" <ron@shell.core.com> wrote in message
news:1163697171.976624.117870@k70g2000cwa.googlegroups.com...
Quote:
monty1945@lycos.com wrote:
Wouldn't it be nice if they informed the public that a diet very low in
unsaturated fatty acids will result in no COX- 2 activity?
COX-2 is essential for fracture healing, so it may not be desirable to
suppress COX-2 activity.
--
Ron
Gee, you're not supposed to come up with a truth. |
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| Vernon |
| Posted: Thu Nov 16, 2006 4:43 pm |
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Guest
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Cox-2 is "natural"
Coconut oil is unique.
Not eating meat is unnatural.
<monty1945@lycos.com> wrote in message
news:1163702948.541731.178000@k70g2000cwa.googlegroups.com...
Quote: COX-2 is essential for fracture healing, so it may not be desirable to
suppress COX-2 activity.
Care to cite some on point evidence for this claim, Ron?
You do understand that Mead acid cannot be made into COX-2 metabolites,
right?
The 1948 experiment found that rats had no problems on a fat free diet,
one that would also mean no COX-2 activity. I have done this
experiment to myself since 2001, though I eat highly saturated fats
with very little linoleic acid content. I notice that cuts actually
heal much better than before, with no scarring problems.
I will challenge you to an experiment in which rats are fed two
different diets, one that is 30% canola and fish oil, and another that
is 30% coconut oil (my choice). If there is no discernable difference
in healing, or if the coconut fed rats heal better, then you pay for
all expenses. Note that biases must be discarded. It is true that the
AA metabolites are more reactive, and cuts might heal a bit faster, but
this often means scarring, and it has nothing to do with claims of
"essentiality."
As to the use of the term "they." Since just about all the "experts"
in the USA are making these kinds of claims, how could I name them all?
I could say, "everyone except Ray Peat and a few others," but I doubt
that you would be satisfied by this qualification, or would you? The
evidence requires no conspiracy theory, only an advocate who is not
conflicted and who is willing to examine it without any biases.
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| Ron Peterson |
| Posted: Thu Nov 16, 2006 7:52 pm |
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Guest
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Vernon wrote:
Quote: "Ron Peterson" <ron@shell.core.com> wrote in message
news:1163697171.976624.117870@k70g2000cwa.googlegroups.com...
COX-2 is essential for fracture healing, so it may not be desirable to
suppress COX-2 activity.
Gee, you're not supposed to come up with a truth.
It may only apply to rats, the study is in J Bone Miner Res. 2002
Jun;17(6):963-76.
The Drug Safety Journal has an article that states:
"Cyclo-oxygenase (COX) is one of the key enzymes in the biosynthesis of
prostaglandins. Two isoforms of this enzyme COX-1 and COX-2 are known
to exist. Among other functions, prostaglandins play an important role
in the protection of the gastric mucosa and maintenance of renal
function in pathophysiological conditions which would otherwise
threaten it. Conventional nonsteroidal anti-inflammatory drugs (NSAIDs)
block prostaglandin synthesis, resulting in gastric mucosal injury and
renal dysfunction in susceptible individuals. The recent introduction
of selective COX-2 inhibitors, celecoxib and rofecoxib, appear to
induce less gastrointestinal morbidity. Although conclusive data are
still lacking, there is evidence to suggest that COX-2 antagonists may
be capable of causing some of the same renal syndromes seen in
association with the older, less selective NSAIDs."
Returning to the original topic, Science Daily has an article that
states:
"Fatty fish contain large amounts of omega-3 fatty acids--diet-derived
essential fatty acids known to benefit patients with cardiovascular
disease and arthritis. This research group recently identified a new
class of aspirin-triggered bioactive lipids, called resolvins, the
activity of which may in part explain the beneficial effects of omega-3
fatty acids. Resolvins are made from the omega-3 fatty acids by
cellular enzymes and can reduce inflammation in mice. The main
bioactive component of this class of lipids was identified in mice and
named resolvin E1."
--
Ron |
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