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Guest
Posted: Wed Mar 07, 2007 7:11 pm
Quote:
From a recent report:

Researchers " examined animals infused with unsaturated oils and found
that they became insulin resistant without any rise in ceramide..."

That is consistent with the molecular-level evidence. But then: "The
team also found that infusion of lard oil, a saturated fat, caused the
mice to become less responsive to insulin..."

Lard is about 40% saturated these days. It is not a "saturated fat"
to anyone who possesses basic common sense. Why did they refrain from
using fresh coconut oil,which is about 92% saturated fatty acids? If
they had, they could make a much stronger case for the claim that
"saturated fat" causes insulin resistance. There is no insulin
resistance on native peoples who have stayed on their native diets,
which are very rich in coconut. The WHO statistics are there for
everyone to see, including this ignorant "team" of researchers.

Ironically, the title of this report is "Stress-signaling Fat Is A
Culprit Behind Insulin Resistance." The problem is that saturated
fatty acids do not "signal," but need to be changed by the body into
molecules capable of signaling (such as the Mead acid, which is
polyunsaturated) if the person is on a fat free diet for a certain
period of time. Their claim is a scientific impossibility. However,
due to the "loose" nutritional language, they are able to convince
themselves of clearly false notions.
Guest
Posted: Wed Mar 07, 2007 7:15 pm
Source for the quoted passages in the post above:
http://www.sciencedaily.com/releases/2007/03/070307075807.htm

Correction: "native peoples" should be "certain native peoples."
MattLB
Posted: Thu Mar 08, 2007 11:38 am
Guest
On Mar 7, 11:11 pm, monty1...@lycos.com wrote:
Quote:
Researchers " examined animals infused with unsaturated oils and found
that they became insulin resistant without any rise in ceramide..."

That is consistent with the molecular-level evidence.

Can you explain how unsaturated fatty acids cause insulin resistance,
at the molecular level?

Quote:
But then: "The
team also found that infusion of lard oil, a saturated fat, caused the
mice to become less responsive to insulin..."

Lard is about 40% saturated these days. It is not a "saturated fat"
to anyone who possesses basic common sense.

Differences due to use of lard can be attributed to the bit that's
different between lard and soy oil. Since lard has over twice as much
SFA and a sixth of the PUFA of soy oil, either you say it's the
increased SFA to blame, or the decreased PUFA that's to blame. Given
that the latter would destroy your world view, you have to choose the
former.

There is another reason for chooisng the former though: the starting
point for synthesis of ceramide is palmitic acid - a saturated fatty
acid.

Quote:
Ironically, the title of this report is "Stress-signaling Fat Is A
Culprit Behind Insulin Resistance." The problem is that saturated
fatty acids do not "signal,"

Try reading the article. Ceramide is the stress-signalling fat the
title refers to.

Quote:
Their claim is a scientific impossibility.

No, just your usual miscomprehension at work.

Quote:
due to the "loose" nutritional language, they are able to convince
themselves of clearly false notions.

You accuse others of "loose nutritional language"? The mind boggles.

MattLB
Guest
Posted: Thu Mar 08, 2007 5:40 pm
I've refuted these MattLB claims many times in the past. One thing
I'll mention here is that lard oil is actually much more like olive
oil, and so what they have demonstrated is that "monounsaturated fat"
is very bad, at least potentially (depending upon factors such as
antioxidant protection and cooking techniques), which is the exact
opposite of the current nutritional dogma.

Have you decided to take me up on my experimental offer, yet, MattLB?
Or are you just doing your usual "hit and run," industry shill gig?
Guest
Posted: Thu Mar 08, 2007 6:17 pm
"In adults, insulin resistance has been associated with dietary
polyunsaturated fatty acids..."

Source: Diabetologia. 2002 Mar;45(3):349-55.

Try doing some actual research once in a while.
Guest
Posted: Thu Mar 08, 2007 6:30 pm
"We discuss the concept that the two essential (not produced by the
body and obtained exclusively through the diet) polyunsaturated fatty
acid families-n-6 and n-3-may play a role in the pathogenesis of
insulin resistance through inflammatory pathways. Linoleic acid, the
major n-6 fatty acid, is metabolized into pro-inflammatory arachidonic
acid..."

Source: Metabolic Syndrome and Related Disorders. Jun 2004, Vol. 2,
No. 2 : 124 -128.

Now some argue that loading up your body with fish oil will prevent
all the ill effects of arachidonic acid, whereas I avoid all major
sources of PUFAs and see only benefits (for over 5 years now), though
according to the nutritionists, I should have died a while back, after
suffering terrible "deficiency syptoms." If MattLB thinks I am wrong,
he can take me up on my offer:

One group of health adult rats is fed a diet of "normal calories,"
with 30% fat from canola oil and fish oil, bought from a supermarket,
with only basic multi-vitamin and mineral supplement. The other group
is exactly the same, except with 30% coconut oil of my choice
(purchased from a retailer). The coconut oil group should die much
sooner. If it does not, MattLB pays for all expenses. If they do,
then I will pay for all expenses.

I am waiting, MattLB, for you to finally put your assets where your
oral cavity is located.
 
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